By the end of this chapter you'll be able to…

  • 1Describe a fracture completely and in a fixed order before naming it
  • 2Read the fracture pattern as a record of the energy that caused it
  • 3Distinguish primary and secondary bone healing and the constructs that produce each
  • 4Explain why absent callus is normal after one implant and alarming after another
  • 5Apply Perren's strain theory to explain nonunion in small gaps
  • 6Match each implant to the stability and healing environment it creates
  • 7Justify why articular fractures need anatomical reduction but shafts do not
  • 8Classify physeal injuries by Salter-Harris and state the growth risk of each
  • 9Explain why Salter-Harris type V is a retrospective diagnosis
  • 10Explain how a peripheral physeal bar produces progressive angular deformity
  • 11Grade supracondylar humerus fractures and manage the pink pulseless hand
  • 12Distinguish a perfused pulseless hand from true limb ischaemia
  • 13Explain the retrograde blood supply behind the three classic sites of avascular necrosis
  • 14Manage a clinical scaphoid fracture with normal radiographs
  • 15Select between fixation, hemiarthroplasty and total hip arthroplasty in femoral neck fracture
  • 16State the effect of surgical timing on hip fracture mortality
  • 17Recognise the dislocations that are commonly missed and the views that reveal them
  • 18Distinguish hypertrophic from atrophic nonunion and treat each correctly
  • 19Recall the nerve and vessel at risk for each major fracture and dislocation
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Why this chapter matters in NEET PG
Fracture management looks like a catalogue of eponyms and implants, and candidates try to memorise it as one. It is not a catalogue. Two mechanical principles generate almost all of it: a fracture is a soft-tissue injury that happens to include a broken bone, and how it heals is decided by how much movement is allowed at the fracture line. Every implant choice, every failure to unite and every question about callus follows from those two sentences. Clinically the stakes are concrete and often irreversible, since a missed pink pulseless hand, a hip fracture delayed past 48 hours, a scaphoid discharged on a normal radiograph and an unrecognised knee dislocation each produce permanent harm that was avoidable at the first contact.

Fractures & Dislocations

Fracture management looks like a catalogue: hundreds of eponyms, dozens of classifications, and an implant for every bone.

It is not a catalogue. Two principles generate almost all of it, and both are mechanical.

The first is that a fracture is a soft-tissue injury that happens to include a broken bone. The bone is the part visible on a radiograph, but the periosteum, the muscle envelope, the vessels and the nerves determine whether the bone will heal and whether the limb will work afterwards.

The second is that the way a fracture heals is decided by how much movement is allowed at the fracture line. Hold it perfectly still and bone crosses directly with no callus. Hold it loosely and callus forms. Hold it badly and nothing forms at all.

Every implant decision, every "why did this not unite", and a large share of NEET PG's orthopaedics questions follow from those two sentences.

1. Describe Before You Diagnose

Most marks lost in fracture questions are lost before any decision is made, because the fracture was described sloppily.

A complete description answers six things in order: which bone, which part of it, whether the skin is intact, the fracture pattern, the displacement, and whether a joint is involved.

Site is proximal, shaft or distal third, and for articular injuries the specific segment. Pattern is transverse, oblique, spiral, comminuted or segmental. Displacement is described as the position of the distal fragment relative to the proximal one, in four terms: translation, angulation, rotation and shortening.

Why the pattern matters

The pattern is a record of the force that caused it. A transverse fracture means a bending or direct force. A spiral fracture means a twisting force. Comminution means high energy, and high energy means the soft tissues absorbed a great deal of it too.

This is why a comminuted tibial fracture in a road traffic collision is a more dangerous injury than a spiral one at the same level, even when the radiograph looks similar in length of bone involved.

2. Two Ways Bone Heals

Bone is one of the few tissues that repairs itself with the original tissue rather than a scar. Which repair pathway it uses depends on the mechanical environment.

Secondary (indirect) healing happens when there is controlled micromotion. Haematoma organises, inflammation recruits cells, soft callus of cartilage and fibrous tissue bridges the gap, it mineralises into hard callus, and remodelling restores the shape. Callus is visible on radiographs. This is the normal path for a cast, a nail or a bridging plate.

Primary (direct) healing happens only when the fragments are compressed together with no measurable motion. Osteoclasts at the cut ends bore cutting cones straight across the fracture line, and osteoblasts follow laying down new osteon. There is no callus at all.

The clinical consequence is one that catches candidates out.

Absent callus after a compression plate is normal and expected. Absent callus after an intramedullary nail at three months is a warning sign. The same radiograph means opposite things depending on the implant.

Perren's strain theory

Strain is the change in gap length divided by the original gap length. Granulation tissue tolerates strain above 100 per cent, cartilage about 10 per cent, and lamellar bone under 2 per cent.

A tissue cannot form in an environment whose strain exceeds its tolerance. This single rule explains the paradox of the small gap.

A 1 mm gap that moves 1 mm has 100 per cent strain and cannot make bone. A 10 mm gap that moves the same 1 mm has 10 per cent strain and will make cartilage, then callus, then bone. This is why a rigid plate spanning a tiny residual gap in a comminuted fracture fails, while the same fracture nailed with a bigger visible gap unites.

Comminution helps here rather than hurting: many small gaps share the total motion, so each individual gap sees low strain.

3. What Each Implant Actually Does

Implants are not ranked by strength. They are chosen by which healing environment the fracture needs.

ConstructStabilityHealingTypical use
Cast or braceRelativeCallusUndisplaced or stable patterns, most paediatric shafts
Lag screw plus neutralisation plateAbsoluteDirect, no callusSimple oblique or spiral fractures
Compression plateAbsoluteDirect, no callusSimple transverse fractures, forearm
Bridging plate over comminutionRelativeCallusComminuted metaphyseal fractures
Intramedullary nailRelativeCallusFemoral and tibial shafts
External fixatorRelative, adjustableCallusOpen fractures, damage control, infection

Articular fractures are the exception that proves the rule. A joint surface must be reduced anatomically and held with absolute stability, because cartilage cannot remodel a step and a step of more than about 2 mm produces post-traumatic arthritis.

The shaft, meanwhile, tolerates imperfection: it only needs correct length, alignment and rotation.

4. Salter-Harris: The Physis Fails Before the Ligament

In a child the growth plate is the mechanical weak link. The same force that sprains an adult ankle fractures a child's physis, which is why the phrase "children do not sprain, they fracture" is a useful working rule.

The classification runs through the physis and asks where else the line goes.

TypeLine runsFrequencyGrowth risk
IThrough physis onlyAbout 5 per centLow
IIPhysis plus metaphyseal fragment (Thurston Holland)About 75 per cent, commonestLow
IIIPhysis plus epiphysis, into joint6 to 8 per centHigher, articular
IVMetaphysis through physis into epiphysisUncommonHigh, articular
VCrush of the physisRareHighest

Types I and II usually reduce closed and heal in a cast. Types III and IV cross the joint surface as well as the physis, so they need anatomical reduction and internal fixation, generally with screws placed parallel to and not across the physis.

Type V is almost always a retrospective diagnosis. The initial radiograph looks normal or nearly so, and the injury declares itself months later as a growth arrest. This is worth stating explicitly to parents at the first visit for any physeal injury.

The bar and the deformity

Growth arrest happens when a bony bar tethers part of the plate. A central bar shortens the bone symmetrically. A peripheral bar tethers one side while the rest keeps growing, producing progressive angular deformity.

The younger the child, the more growth remains and the worse the eventual deformity, which inverts the usual reassurance that children heal better.

5. Upper Limb Fractures That Are Always Asked

Supracondylar humerus

The commonest elbow fracture in children, typically from a fall on an outstretched hand producing an extension-type injury. Gartland grades it: I undisplaced, II angulated with intact posterior cortex, III completely displaced, IV unstable in both flexion and extension.

Gartland III carries the vascular and neurological risk. The anterior interosseous nerve is the classic injury in extension type; test it by asking the child to make an "OK" sign, which requires flexor pollicis longus and the index flexor digitorum profundus.

The pink pulseless hand is the decision that gets examined. A hand that is warm and well perfused with normal capillary refill but no palpable radial pulse is perfused through collaterals around the elbow. The primary treatment is urgent reduction and percutaneous pinning, after which the pulse commonly returns.

A hand that is pale, cold and poorly perfused is a true ischaemic emergency and needs vascular exploration. Series of Gartland III fractures report acute ischaemia or a pink pulseless hand in roughly one in six, so this is not a rare scenario.

Do not confuse either with the late catastrophe: Volkmann ischaemic contracture follows unrecognised forearm compartment syndrome, and pain on passive extension of the fingers is its earliest sign.

Scaphoid

The scaphoid is supplied retrograde by the dorsal carpal branch of the radial artery, entering distally. A waist or proximal pole fracture therefore devascularises the proximal fragment, and the more proximal the fracture, the higher the risk of avascular necrosis and nonunion.

A patient with anatomical snuffbox tenderness and a normal radiograph has a clinical scaphoid fracture until proven otherwise. Immobilise and image again. MRI is the reference standard for occult fracture, with sensitivity of about 95 to 100 per cent and specificity near 100 per cent, and early MRI within one to three days is now the recommended pathway where available.

Untreated, the sequence is nonunion, then carpal collapse, then scaphoid nonunion advanced collapse arthritis in a young hand.

Distal radius

Colles is dorsal displacement of the distal radius, classically in an older person after a fall on an outstretched hand, giving the dinner fork deformity. Smith is the volar equivalent. Barton is an intra-articular shear with subluxation of the carpus.

The complication to know is median nerve compression, either immediate from displacement or late from callus. Extensor pollicis longus rupture is the delayed tendon complication, characteristically after an undisplaced fracture rather than a displaced one, because the intact retinacular sheath compromises the tendon's blood supply.

The two forearm eponyms

Monteggia is a proximal ulnar fracture with radial head dislocation. Galeazzi is a distal radial fracture with distal radioulnar joint disruption.

The rule behind both is that the radius and ulna form a ring with the two joints. A ring cannot break in only one place, so a single-bone forearm fracture with any shortening or angulation obliges you to radiograph both the elbow and the wrist.

6. The Hip Fracture Is a Medical Emergency

Femoral neck fractures are intracapsular. The blood supply to the head runs retrograde through the retinacular vessels of the medial femoral circumflex artery, so a displaced neck fracture may destroy it. Intertrochanteric fractures are extracapsular, heal reliably, and are fixed rather than replaced.

That anatomical difference produces the treatment split.

FracturePatientTreatment
Undisplaced neckAnyInternal fixation with screws or sliding hip screw
Displaced neckOlder, lower demandHemiarthroplasty
Displaced neckOlder, active, independentTotal hip arthroplasty
Displaced neckYoungUrgent anatomical reduction and fixation, salvage the head
IntertrochantericAnySliding hip screw or cephalomedullary nail

The evidence supports the split. In displaced fractures, reoperation rates after internal fixation run far higher than after arthroplasty, and in the FAITH trial 18 per cent of fixed patients converted to arthroplasty within 24 months.

Timing is the modifiable variable that changes mortality. Meta-analysis of over 190,000 patients found surgery within 48 hours associated with roughly 20 per cent lower one-year mortality. The fracture itself is survivable; the immobility, delirium, pneumonia and pressure injury that accumulate while waiting are what kill.

This is why hip fracture is now managed as a time-critical pathway with orthogeriatric co-care rather than as an elective list item.

7. Dislocations: Reduce Early, Then Ask Why

A dislocation compresses vessels, stretches nerves and starves cartilage of nutrition. The urgency is not the pain, it is the tissue clock.

Anterior shoulder dislocation is the commonest major joint dislocation. The axillary nerve is at risk, tested by sensation over the deltoid badge area before and after reduction. The structural lesions are the Bankart lesion of the anteroinferior labrum and the Hill-Sachs impaction on the posterolateral humeral head.

Age drives prognosis. Young patients, particularly athletes in contact sport, have recurrence rates reported between about two-thirds and over 90 per cent after a first dislocation, which is why arthroscopic stabilisation is discussed early rather than after the fifth episode.

Posterior shoulder dislocation is the one that is missed, classically after a seizure or electric shock. The arm is held internally rotated, the anteroposterior film shows the light bulb sign, and the axillary or scapular Y view makes the diagnosis. Any shoulder radiograph without a second orthogonal view is an incomplete examination.

Posterior hip dislocation accounts for the large majority of hip dislocations and typically follows a dashboard injury with the hip flexed and adducted. The limb lies shortened, flexed, adducted and internally rotated. The sciatic nerve, especially its peroneal division, is at risk.

Reduction should be achieved within about six hours, because the rate of avascular necrosis of the femoral head rises steeply with delay. A post-reduction CT is standard to look for the acetabular wall fracture and any incarcerated fragment.

8. When Healing Fails

Delayed union is a fracture taking longer than expected for its site. Nonunion is a fracture that has stopped trying, defined clinically and radiologically rather than purely by a date.

Nonunions divide by their biology, and the division dictates treatment.

Hypertrophic nonunion shows abundant callus with a persistent line, often described as an elephant foot. The biology is good and the mechanics are bad: it is moving too much. Stabilise it and it unites.

Atrophic nonunion shows no callus and tapered bone ends. The mechanics may be adequate but the biology has failed, from stripped periosteum, infection, poor blood supply, smoking or diabetes. It needs debridement, bone graft and correction of the underlying cause, not simply a stronger implant.

The reliable framework is the diamond concept: union requires mechanical stability, osteogenic cells, an osteoconductive scaffold and osteoinductive signals. Ask which of the four is missing before choosing the revision.

Malunion and other outcomes

Malunion is union in an unacceptable position. Rotational malunion of the femur is the classic example, because rotation is the deformity that does not remodel at any age and is easy to miss intraoperatively.

Complex regional pain syndrome deserves separate mention: pain disproportionate to the injury with vasomotor, sudomotor and trophic changes, most often after distal radius fracture. Early mobilisation and analgesia matter more than any single drug.

Fat embolism syndrome appears 24 to 72 hours after long bone or pelvic fracture with the triad of hypoxia, neurological change and a petechial rash in the axilla, conjunctiva and upper chest. Early fracture fixation reduces the risk; treatment is supportive.

9. High-Yield Associations

Fracture or dislocationComplication to expect
Femoral neck, displacedAVN of femoral head
Scaphoid waist or proximal poleAVN of proximal pole, nonunion
Talar neckAVN of talar body
Humeral shaft, distal thirdRadial nerve palsy, wrist drop
Supracondylar humerus, extensionAnterior interosseous nerve, brachial artery
Medial epicondyleUlnar nerve
Surgical neck of humerus, anterior shoulder dislocationAxillary nerve
Posterior hip dislocationSciatic nerve, AVN of femoral head
Knee dislocationPopliteal artery, common peroneal nerve
Distal radiusMedian nerve, late EPL rupture

Knee dislocation deserves emphasis. It may reduce spontaneously before arrival, so a knee that is grossly unstable in multiple planes after trauma should be treated as a dislocation even if it looks normal on the film. Popliteal artery injury is the limb-threatening complication and mandates vascular assessment, with ankle-brachial index and CT angiography as indicated.

10. Worked Examples

Example 1. A 7-year-old falls on an outstretched hand. The elbow is swollen, radiographs show a completely displaced supracondylar fracture, and the hand is warm and pink with brisk capillary refill but no radial pulse. What is the immediate management?

Urgent closed reduction and percutaneous pinning under anaesthesia. The hand is perfused through collaterals, and the pulse usually returns once the fracture is reduced and the brachial artery is no longer tented. Vascular exploration is reserved for a hand that is pale, cold or shows deteriorating perfusion after reduction. Document anterior interosseous nerve function before and after.

Example 2. A comminuted distal femoral fracture is treated with a bridging plate. At 12 weeks there is abundant callus but the patient still has pain on weight bearing. A second patient with a simple transverse femoral fracture treated by compression plating shows no callus at all at 12 weeks and is comfortable. Which one is failing?

Neither is failing on the callus finding. The bridging plate provides relative stability, so callus is the expected and correct appearance. The compression plate provides absolute stability, so direct healing with no callus is expected. Callus after a compression plate would suggest the construct is loose. Judge each construct against the healing pathway it was designed to produce.

Example 3. A 22-year-old sustains a spiral tibial fracture. It is fixed with a rigid plate leaving a 1 mm residual gap. At six months there is no union and no callus. Explain the mechanical error.

Perren's strain theory. A tiny gap subjected to any residual micromotion generates very high strain, because strain is motion divided by gap length. Above roughly 2 per cent, lamellar bone cannot form, and above about 10 per cent even cartilage cannot. The gap was too small to tolerate the motion that the construct still permitted. Either true compression with no motion, or a larger working length allowing callus, would have united it.

Example 4. A 78-year-old woman with a displaced femoral neck fracture is admitted on Friday evening. She lives independently and walks to the market daily. The list is full until Monday. What are the two key decisions?

First, operate within 48 hours: delay beyond that is associated with roughly 20 per cent higher one-year mortality, so she should be prioritised rather than deferred. Second, because she is active and independent with a displaced intracapsular fracture, total hip arthroplasty gives better function and lower reoperation than internal fixation or hemiarthroplasty in this group.

Example 5. A 12-year-old has an ankle injury with tenderness over the distal tibial physis and normal radiographs. He is treated in a cast and recovers. Eighteen months later the ankle is progressively angulating into varus. What happened?

A Salter-Harris type V crush injury, diagnosed retrospectively. The physis was compressed at the time of injury without a visible fracture line, a bony bar formed peripherally on the medial side, and the tethered medial physis stopped growing while the lateral side continued, producing progressive varus. This is why every physeal injury warrants a warning about growth and follow-up rather than discharge at cast removal.

Summary

A fracture is a soft-tissue injury that includes a broken bone, and the soft tissues decide the outcome.

Describe before diagnosing: bone, site, skin, pattern, displacement, joint involvement.

Bone heals two ways. Absolute stability gives direct healing with no callus; relative stability gives callus. The implant chosen determines which appearance is normal.

Perren's strain theory explains failures: strain is motion divided by gap length, so small gaps are dangerous, and lamellar bone needs strain under 2 per cent.

Joint surfaces need anatomical reduction and absolute stability; shafts need only length, alignment and rotation.

In children the physis fails before the ligament. Salter-Harris II is commonest; III and IV cross the joint and need fixation; V is diagnosed retrospectively as growth arrest.

Supracondylar Gartland III: reduce and pin urgently, and distinguish the pink pulseless hand from true ischaemia.

Retrograde blood supply explains the AVN triad: femoral head, scaphoid proximal pole, talar body.

Displaced femoral neck fractures in older active patients do better with total hip arthroplasty, and surgery within 48 hours lowers one-year mortality by about a fifth.

Reduce dislocations early: six hours for the hip before AVN risk climbs, and never accept a single view of a shoulder.

Hypertrophic nonunion is a mechanical problem, atrophic nonunion a biological one. Fix the one that is actually missing.

Key formulas & results

Everything to memorise for the exam hall, in one card. Screenshot this for revision.

The organising tool
A FRACTURE IS A SOFT-TISSUE INJURY THAT HAPPENS TO INCLUDE A BROKEN BONE, AND HOW IT HEALS IS DECIDED BY HOW MUCH MOVEMENT IS ALLOWED AT THE FRACTURE LINE.
THE BONE IS THE PART VISIBLE ON THE RADIOGRAPH, BUT THE PERIOSTEUM, MUSCLE ENVELOPE, VESSELS AND NERVES DECIDE WHETHER IT UNITES AND WHETHER THE LIMB WORKS AFTERWARDS.
Perren's strain theory
STRAIN = CHANGE IN GAP LENGTH DIVIDED BY ORIGINAL GAP LENGTH. GRANULATION TISSUE TOLERATES OVER 100 PER CENT, CARTILAGE ABOUT 10 PER CENT, LAMELLAR BONE UNDER 2 PER CENT.
A TISSUE CANNOT FORM WHERE STRAIN EXCEEDS ITS TOLERANCE. THIS IS THE SINGLE EQUATION BEHIND MOST MECHANICAL NONUNIONS.
The paradox of the small gap
A 1 MM GAP MOVING 1 MM HAS 100 PER CENT STRAIN AND CANNOT MAKE BONE. A 10 MM GAP MOVING 1 MM HAS 10 PER CENT STRAIN AND WILL MAKE CALLUS.
SMALL GAPS ARE MORE DANGEROUS THAN LARGE ONES UNLESS MOTION IS ABOLISHED COMPLETELY. COMMINUTION HELPS BECAUSE MANY GAPS SHARE THE TOTAL MOTION.
Two healing pathways
ABSOLUTE STABILITY GIVES PRIMARY HEALING BY CUTTING CONES WITH NO CALLUS. RELATIVE STABILITY GIVES SECONDARY HEALING THROUGH SOFT THEN HARD CALLUS.
NEITHER IS SUPERIOR. THEY ARE DIFFERENT ANSWERS TO DIFFERENT MECHANICAL QUESTIONS.
Reading callus against the implant
NO CALLUS AFTER A COMPRESSION PLATE IS EXPECTED. NO CALLUS AFTER AN INTRAMEDULLARY NAIL AT THREE MONTHS IS A WARNING. CALLUS AFTER A COMPRESSION PLATE SUGGESTS THE CONSTRUCT IS LOOSE.
THE SAME RADIOGRAPH MEANS OPPOSITE THINGS DEPENDING ON WHAT THE IMPLANT WAS DESIGNED TO DO.
Complete fracture description
WHICH BONE, WHICH PART, SKIN INTACT OR NOT, PATTERN, DISPLACEMENT, JOINT INVOLVEMENT.
DISPLACEMENT IS THE POSITION OF THE DISTAL FRAGMENT RELATIVE TO THE PROXIMAL ONE, IN FOUR TERMS: TRANSLATION, ANGULATION, ROTATION AND SHORTENING.
Pattern as energy record
TRANSVERSE MEANS BENDING OR DIRECT FORCE. SPIRAL MEANS TWISTING. COMMINUTED OR SEGMENTAL MEANS HIGH ENERGY.
HIGH ENERGY MEANS THE SOFT TISSUES ABSORBED A GREAT DEAL OF IT TOO, WHICH IS WHY TWO SIMILAR-LOOKING FILMS CAN BE VERY DIFFERENT INJURIES.
Shaft versus joint
A SHAFT NEEDS ONLY CORRECT LENGTH, ALIGNMENT AND ROTATION. AN ARTICULAR SURFACE NEEDS ANATOMICAL REDUCTION AND ABSOLUTE STABILITY.
CARTILAGE CANNOT REMODEL A STEP, AND A STEP OVER ABOUT 2 MM PRODUCES POST-TRAUMATIC ARTHRITIS.
Salter-Harris frequency and risk
TYPE I PHYSIS ONLY, ABOUT 5 PER CENT. TYPE II PHYSIS PLUS METAPHYSIS, ABOUT 75 PER CENT AND COMMONEST. TYPE III PHYSIS PLUS EPIPHYSIS, 6 TO 8 PER CENT. TYPE IV CROSSES BOTH. TYPE V IS A CRUSH.
GROWTH RISK RISES DOWN THE LIST. TYPES III AND IV ALSO CROSS THE JOINT SURFACE, SO THEY CARRY TWO SEPARATE REASONS FOR ANATOMICAL FIXATION.
Children fracture rather than sprain
THE PHYSIS IS THE MECHANICAL WEAK LINK, SO THE FORCE THAT SPRAINS AN ADULT ANKLE FRACTURES A CHILD'S GROWTH PLATE.
A TENDER PHYSIS WITH A NORMAL FILM IN A CHILD IS TREATED AS A FRACTURE, NOT AS A LIGAMENT INJURY.
Physeal bar geometry
A CENTRAL BAR SHORTENS THE BONE SYMMETRICALLY. A PERIPHERAL BAR TETHERS ONE SIDE WHILE THE REST KEEPS GROWING, PRODUCING PROGRESSIVE ANGULAR DEFORMITY.
THE YOUNGER THE CHILD, THE MORE GROWTH REMAINS AND THE WORSE THE FINAL DEFORMITY, WHICH INVERTS THE USUAL REASSURANCE THAT CHILDREN HEAL BETTER.
Gartland grading
I UNDISPLACED. II ANGULATED WITH INTACT POSTERIOR CORTEX. III COMPLETELY DISPLACED. IV UNSTABLE IN BOTH FLEXION AND EXTENSION.
GRADE III CARRIES THE VASCULAR AND NEUROLOGICAL RISK, WITH ACUTE ISCHAEMIA OR A PINK PULSELESS HAND IN ROUGHLY ONE IN SIX IN PUBLISHED SERIES.
The pink pulseless hand
WARM, PINK, BRISK CAPILLARY REFILL, NO RADIAL PULSE MEANS PERFUSION THROUGH COLLATERALS. URGENT REDUCTION AND PINNING FIRST, AND THE PULSE USUALLY RETURNS.
PALE, COLD AND POORLY PERFUSED IS A DIFFERENT INJURY AND NEEDS VASCULAR EXPLORATION. THE TWO WORDS PINK AND PALE CARRY THE ENTIRE DECISION.
Anterior interosseous nerve test
ASK THE CHILD TO MAKE AN OK SIGN, WHICH REQUIRES FLEXOR POLLICIS LONGUS AND THE INDEX FLEXOR DIGITORUM PROFUNDUS.
IT IS A PURELY MOTOR NERVE, SO THERE IS NO SENSORY DEFICIT TO PROMPT YOU. DOCUMENT IT BEFORE AND AFTER REDUCTION.
Retrograde supply and avascular necrosis
FEMORAL HEAD THROUGH RETINACULAR VESSELS OF THE MEDIAL FEMORAL CIRCUMFLEX. SCAPHOID PROXIMAL POLE THROUGH THE DORSAL CARPAL BRANCH ENTERING DISTALLY. TALAR BODY THROUGH THE NECK.
IN ALL THREE THE FRACTURE LIES BETWEEN THE VESSEL AND THE FRAGMENT IT SUPPLIES, SO THE MORE PROXIMAL THE SCAPHOID FRACTURE AND THE MORE DISPLACED THE NECK, THE HIGHER THE RISK.
Intracapsular versus extracapsular hip
FEMORAL NECK IS INTRACAPSULAR AND THREATENS THE HEAD'S BLOOD SUPPLY. INTERTROCHANTERIC IS EXTRACAPSULAR, HEALS RELIABLY AND IS FIXED RATHER THAN REPLACED.
THIS ONE ANATOMICAL LINE GENERATES THE ENTIRE TREATMENT TABLE FOR HIP FRACTURE.
Hip fracture timing
SURGERY WITHIN 48 HOURS IS ASSOCIATED WITH ROUGHLY 20 PER CENT LOWER ONE-YEAR MORTALITY IN META-ANALYSIS OF OVER 190,000 PATIENTS.
THE FRACTURE IS SURVIVABLE. THE IMMOBILITY, DELIRIUM, PNEUMONIA AND PRESSURE INJURY THAT ACCUMULATE WHILE WAITING ARE WHAT KILL.
The forearm ring
THE RADIUS AND ULNA WITH THEIR TWO JOINTS FORM A RING, AND A RING CANNOT BREAK IN ONLY ONE PLACE.
A SINGLE-BONE FOREARM FRACTURE WITH SHORTENING OR ANGULATION OBLIGES RADIOGRAPHS OF BOTH ELBOW AND WRIST. MONTEGGIA IS PROXIMAL ULNA WITH RADIAL HEAD DISLOCATION, GALEAZZI IS DISTAL RADIUS WITH DISTAL RADIOULNAR DISRUPTION.
Hip dislocation clock
REDUCE A DISLOCATED HIP WITHIN ABOUT SIX HOURS, BECAUSE THE RATE OF AVASCULAR NECROSIS RISES STEEPLY WITH DELAY.
POSTERIOR DISLOCATION IS THE LARGE MAJORITY, WITH THE LIMB SHORTENED, FLEXED, ADDUCTED AND INTERNALLY ROTATED, AND THE SCIATIC NERVE AT RISK.
Nonunion biology
HYPERTROPHIC WITH ABUNDANT CALLUS MEANS GOOD BIOLOGY AND BAD MECHANICS: STABILISE IT. ATROPHIC WITH NO CALLUS MEANS THE BIOLOGY HAS FAILED: DEBRIDE, GRAFT AND CORRECT THE CAUSE.
A STRONGER IMPLANT ALONE FIXES THE FIRST AND NOT THE SECOND, WHICH IS WHY THE DISTINCTION IS MADE BEFORE THE REVISION IS PLANNED.
The diamond concept
UNION REQUIRES MECHANICAL STABILITY, OSTEOGENIC CELLS, AN OSTEOCONDUCTIVE SCAFFOLD AND OSTEOINDUCTIVE SIGNALS.
ASK WHICH OF THE FOUR IS MISSING BEFORE CHOOSING THE REVISION STRATEGY.
Fat embolism syndrome
24 TO 72 HOURS AFTER LONG BONE OR PELVIC FRACTURE, WITH HYPOXIA, NEUROLOGICAL CHANGE AND A PETECHIAL RASH IN THE AXILLA, CONJUNCTIVA AND UPPER CHEST.
EARLY FRACTURE FIXATION REDUCES THE RISK AND TREATMENT IS SUPPORTIVE. THE TIMING WINDOW IS WHAT SEPARATES IT FROM PULMONARY EMBOLISM IN A STEM.
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Traps NEET PG sets — and how to dodge them

These are the exact option-traps and misreads that cost marks under negative marking.

WATCH OUT
Treating a fracture as a bone problem rather than a soft-tissue injury
The periosteum, muscle envelope, vessels and nerves decide union and function, and the fracture pattern is a record of how much energy those tissues absorbed. A comminuted tibial fracture from a collision is a far more dangerous injury than a spiral one of the same length.
WATCH OUT
Reading absent callus as failure regardless of the implant
A compression plate produces absolute stability and direct healing, so no callus is the expected appearance. Callus after a compression plate suggests the construct is loose, while absent callus after a nail at three months is the finding that should worry you.
WATCH OUT
Assuming a smaller gap always heals better
Strain is motion divided by gap length, so a 1 mm gap moving 1 mm has 100 per cent strain and cannot form bone, while a 10 mm gap moving the same amount has 10 per cent and will form callus. Either abolish the motion completely or allow a working length.
WATCH OUT
Choosing implants by strength rather than by the healing environment needed
Bridging plates, nails and external fixators deliberately provide relative stability so that callus can form, while lag screws and compression plates deliberately abolish motion. The question is which pathway the fracture needs, not which construct is stiffest.
WATCH OUT
Accepting the same reduction standard for shafts and joint surfaces
A shaft needs only length, alignment and rotation because it remodels and functions as a lever. An articular surface cannot remodel a step, so a step over roughly 2 mm produces post-traumatic arthritis and demands anatomical reduction with absolute stability.
WATCH OUT
Diagnosing a sprain in a child with a tender growth plate
The physis is weaker than the surrounding ligament, so the force that sprains an adult ankle fractures a child's plate. Tenderness over the physis with a normal radiograph is managed as a Salter-Harris injury until follow-up says otherwise.
WATCH OUT
Discharging a physeal injury at cast removal
Type V crush injuries are invisible initially and declare themselves months later as growth arrest, and a peripheral bar produces progressive angulation rather than simple shortening. Every physeal injury warrants an explicit warning about growth and planned follow-up.
WATCH OUT
Exploring the vessels in every pulseless supracondylar fracture
A warm pink hand with brisk capillary refill is perfused through collaterals, and urgent reduction with percutaneous pinning usually restores the pulse. Exploration belongs to the pale, cold, poorly perfused hand and to perfusion that deteriorates after reduction.
WATCH OUT
Missing the anterior interosseous nerve because sensation is normal
It is a purely motor nerve, so there is no sensory deficit to prompt testing. Ask the child to make an OK sign, which requires flexor pollicis longus and the index flexor digitorum profundus, and record the result before and after reduction.
WATCH OUT
Discharging a snuffbox-tender wrist on a normal radiograph
Occult scaphoid fracture is common and the initial film is frequently normal. Immobilise and reimage, with MRI as the reference standard at sensitivity of roughly 95 to 100 per cent, because the untreated sequence is nonunion, carpal collapse and arthritis in a young hand.
WATCH OUT
Offering hemiarthroplasty to every older patient with a displaced neck fracture
Independent, active patients do better with total hip arthroplasty, which gives superior function and markedly lower reoperation than internal fixation in displaced intracapsular fractures. Hemiarthroplasty suits lower-demand patients, and young patients get urgent reduction and fixation to salvage the head.
WATCH OUT
Treating hip fracture surgery as an elective list item
Delay beyond 48 hours is associated with roughly 20 per cent higher one-year mortality, and the deaths come from immobility, delirium, pneumonia and pressure injury rather than from the fracture. Modern practice runs a time-critical pathway with orthogeriatric co-care.
WATCH OUT
Accepting a single anteroposterior view of an injured shoulder
Posterior dislocation after seizure or electric shock shows only a light bulb sign on that view and is the classic miss. An axillary or scapular Y view is required, and a shoulder radiograph without a second orthogonal projection is an incomplete examination.
WATCH OUT
Reassuring a young patient after a first shoulder dislocation
Recurrence in young patients, particularly contact athletes, is reported between roughly two-thirds and over 90 per cent after a first event. That is why arthroscopic stabilisation is discussed early rather than after repeated dislocations have damaged the glenoid rim.
WATCH OUT
Dismissing a knee that looks normal after high-energy trauma
A knee dislocation often reduces spontaneously before arrival, so gross multiplanar instability after trauma is treated as a dislocation regardless of the film. Popliteal artery injury is limb-threatening and mandates vascular assessment with ankle-brachial index and imaging as indicated.
WATCH OUT
Revising an atrophic nonunion with a bigger plate
Abundant callus with a persistent line is a mechanical problem that stabilisation cures, while absent callus with tapered ends is a biological failure from stripped periosteum, infection, poor supply, smoking or diabetes. The second needs debridement, graft and correction of the cause.

Exam-pattern practice

PYQ-style questions with full solutions. Work through them as a readiness check — mark yourself honestly and get your gap report at the end.

Readiness check

Are you exam-ready for "Fractures & Dislocations"?

9 problems from this chapter. Try each one, reveal the worked solution, mark yourself honestly — get your gap report at the end.

9 questions~6 min

5-minute revision

The whole chapter, distilled. Read this the night before the exam.

  • A fracture is a soft-tissue injury that includes a broken bone.
  • Describe before diagnosing: bone, site, skin, pattern, displacement, joint.
  • Displacement is the distal fragment relative to the proximal one.
  • Four displacement terms: translation, angulation, rotation, shortening.
  • Transverse means bending, spiral means twisting, comminuted means high energy.
  • High energy on the film means high energy through the soft tissues.
  • Secondary healing gives callus; primary healing gives none.
  • Primary healing needs compression and cutting cones.
  • No callus after a compression plate is normal.
  • No callus after a nail at three months is a warning.
  • Callus after a compression plate suggests a loose construct.
  • Strain is motion divided by gap length.
  • Lamellar bone needs strain under 2 per cent, cartilage about 10 per cent.
  • A 1 mm gap moving 1 mm has 100 per cent strain.
  • Comminution shares motion between many gaps and lowers strain in each.
  • Lag screw and compression plate give absolute stability.
  • Nail, bridging plate and external fixator give relative stability.
  • Articular surfaces need anatomical reduction and absolute stability.
  • A step over about 2 mm in cartilage produces post-traumatic arthritis.
  • Shafts need only length, alignment and rotation.
  • Rotational malunion does not remodel at any age.
  • In children the physis fails before the ligament.
  • Salter-Harris II is commonest at about 75 per cent.
  • The Thurston Holland fragment is the metaphyseal piece in type II.
  • Types I and II usually reduce closed and heal in a cast.
  • Types III and IV cross the joint and need anatomical fixation.
  • Screws for physeal fractures run parallel to the physis, not across it.
  • Type V is a crush and is diagnosed retrospectively as growth arrest.
  • A central physeal bar shortens; a peripheral bar angulates.
  • Younger children have more growth left and worse eventual deformity.
  • Supracondylar humerus is the commonest paediatric elbow fracture.
  • Gartland III is completely displaced and carries the vascular risk.
  • Anterior interosseous nerve is tested with the OK sign.
  • The anterior interosseous nerve is purely motor, so sensation is normal.
  • Pink pulseless hand: reduce and pin urgently, pulse usually returns.
  • Pale cold hand: true ischaemia, explore the vessels.
  • Volkmann contracture follows missed forearm compartment syndrome.
  • Pain on passive finger extension is the earliest compartment sign.
  • Scaphoid supply is retrograde from the dorsal carpal branch.
  • Proximal pole scaphoid fractures carry the highest avascular necrosis risk.
  • Snuffbox tenderness with a normal film is a fracture until disproved.
  • MRI is the reference standard for occult scaphoid fracture.
  • Colles is dorsal, Smith is volar, Barton is intra-articular shear.
  • Late EPL rupture follows undisplaced distal radius fractures.
  • Complex regional pain syndrome most often follows distal radius fracture.
  • Monteggia: proximal ulna fracture with radial head dislocation.
  • Galeazzi: distal radius fracture with distal radioulnar disruption.
  • A single-bone forearm fracture demands elbow and wrist films.
  • Femoral neck is intracapsular; intertrochanteric is extracapsular.
  • Displaced neck fracture in an active older patient: total hip arthroplasty.
  • Displaced neck fracture in a young patient: urgent reduction and fixation.
  • Intertrochanteric fractures are fixed, not replaced.
  • Hip fracture surgery within 48 hours lowers one-year mortality by about a fifth.
  • Anterior shoulder dislocation risks the axillary nerve.
  • Bankart is the labral lesion, Hill-Sachs the humeral impaction.
  • Young first-time dislocators recur in two-thirds or more.
  • Posterior shoulder dislocation follows seizure or electric shock.
  • The light bulb sign needs an axillary or scapular Y view to confirm.
  • Posterior hip dislocation: shortened, flexed, adducted, internally rotated.
  • Reduce a dislocated hip within about six hours.
  • Knee dislocation may self-reduce; suspect popliteal artery injury.
  • Hypertrophic nonunion is mechanical: stabilise it.
  • Atrophic nonunion is biological: debride, graft, correct the cause.
  • The diamond concept: stability, cells, scaffold, signals.
  • Fat embolism appears 24 to 72 hours after long bone fracture.
  • The fat embolism triad is hypoxia, confusion and petechial rash.

NEET PG question blueprint

How this topic is asked, tier by tier — so you can prep to the pattern.

Typical weightage: Each NEET PG question is worth +4/-1; fractures and dislocations contribute 6-8 questions per attempt, the largest single orthopaedic block, and overlap with Anatomy, Radiology and Surgery

Question styleMarks eachTypical countWhat it tests
Healing and stability4~1Primary versus secondary healing, Perren's strain theory and matching implant to healing pathway
Describing the fracture4~1Pattern, displacement terms and reading energy from the radiograph
Paediatric physeal injury4~1Salter-Harris types, frequency, growth risk and the retrospective type V
Supracondylar humerus4~1Gartland grading, anterior interosseous nerve testing and the pink pulseless hand decision
Hip fracture4~1Intracapsular versus extracapsular anatomy, implant selection by age and demand, and the 48-hour timing evidence
Dislocations4~1Anterior and posterior shoulder, posterior hip, knee dislocation and the views that reveal them
Nerve and vascular associations4~1The fracture-to-nerve table and the three retrograde avascular necrosis sites
Nonunion and complications4~1Hypertrophic versus atrophic nonunion, malunion, fat embolism and complex regional pain syndrome

Exam-hall strategy

Battle-tested tips from mentors and toppers for this topic under the sectional clock.

  1. Identify whether the construct in the stem gives absolute or relative stability before judging the radiograph.
  2. For any nonunion question, first classify it as hypertrophic or atrophic; the treatment follows automatically.
  3. In paediatric stems, check whether the physis is involved and whether the joint surface is crossed.
  4. For a pulseless hand, read the two words describing colour and temperature; they carry the whole answer.
  5. For femoral neck stems, extract displacement, age and premorbid activity before choosing an implant.
  6. When a nerve deficit is offered, match it to the fracture site rather than to the symptom alone.
  7. Suspect a missed dislocation whenever only one radiographic view is mentioned.
  8. With NEET PG's +4/-1 marking, the Salter-Harris table, the nerve associations and the avascular necrosis sites are high-certainty recall worth banking early.
  9. Under the 5-group, 42-minute time-bound format, clear those recall items fast and spend the remaining time on the stability and hip fracture management stems, since a closed group cannot be reopened.

Beyond the exam

Where this skill shows up in the job you're competing for — and in life.

Prioritising the frail hip fracture onto today's list

Treating time to theatre as a clinical variable rather than a scheduling one is the single change that most reduces one-year mortality after hip fracture, and it requires optimisation to run in parallel with booking rather than before it.

Reducing before imaging in the ischaemic elbow

Recognising that a displaced supracondylar fracture is tenting the brachial artery converts a vascular emergency into a fracture problem, and the reduction that restores the pulse also removes the need for exploration.

Casting the normal-looking wrist

Immobilising and reimaging a snuffbox-tender wrist despite a clear radiograph prevents the slow progression through scaphoid nonunion and carpal collapse into arthritis in a hand that has decades of work ahead of it.

Choosing the construct for the biology available

Matching stability to the healing pathway, absolute for a simple joint fracture and relative for a comminuted shaft, avoids the commonest cause of avoidable nonunion, which is a rigid implant across a gap too small to tolerate motion.

Where else this topic is tested

Prepare once, score in every exam that asks it.

FMGE / NExTVery high overlap — supracondylar humerus, Salter-Harris, hip fracture management and the nerve associations are examined at identical depth
USMLE Step 2 CKHigh overlap — hip fracture timing, scaphoid imaging, dislocation complications and compartment syndrome are shared, with less emphasis on implant mechanics
MS Orthopaedics and DNB entranceFoundational — assumed working knowledge, with AO classification, biomechanics of fixation and reconstruction options examined far more deeply

Questions aspirants ask

Pulled from the Q&A community and mentor sessions.

Because healing responds to strain, not to distance. Strain is the change in gap length divided by the original gap length, so the same physical movement produces wildly different strains depending on how much gap there is to absorb it. One millimetre of motion across a one millimetre gap is 100 per cent strain, an environment in which only granulation tissue can survive. The same one millimetre of motion across a ten millimetre gap is 10 per cent strain, which cartilage tolerates, so soft callus forms, mineralises and bridges. The tissue hierarchy is fixed: granulation tolerates over 100 per cent, cartilage roughly 10 per cent, lamellar bone under 2 per cent. This is why comminution is mechanically helpful rather than harmful once the fracture is stabilised with relative stability, since many small gaps divide the total motion between them and each individual gap sees a strain it can work with. It also explains the classic failure of a rigid plate applied across a simple fracture with a tiny residual gap: the surgeon achieved neither true compression nor a workable gap, and the fracture is left in the one region of the strain curve where nothing can form.

Because callus is evidence of a particular healing pathway, not evidence of healing in general. Bone has two repair routes. Under absolute stability with interfragmentary compression, osteoclasts drill cutting cones directly across the fracture line and osteoblasts follow, laying down new osteons that span the break. Nothing needs to bridge on the outside, so no callus appears. Under relative stability with controlled micromotion, the fracture heals through haematoma, soft callus, hard callus and remodelling, and the callus is visible. So the correct interpretation depends entirely on what the surgeon set out to create. After a compression plate or a lag screw with a neutralisation plate, absent callus is the intended result and its appearance would suggest the construct has loosened. After an intramedullary nail or a bridging plate, callus is the intended result and its absence at three months means the fracture is not progressing. The examinable principle is that a radiograph is judged against the mechanical environment of the implant, never in isolation.

Because the pulse and the perfusion are answering different questions, and only perfusion determines whether tissue is dying. The elbow has a rich collateral network, and in a displaced supracondylar fracture the brachial artery is most often tented, kinked or in spasm over the proximal fragment rather than transected. Flow through the collaterals maintains a warm hand with brisk capillary refill even though nothing is palpable at the wrist. Reducing and pinning the fracture removes the mechanical cause, and in the large majority the pulse returns on the table or within hours. Exploring first would mean operating on a vessel that did not need repair while the fracture stayed displaced. The reasoning inverts completely if the hand is pale, cold, mottled or has sluggish refill, because that indicates the collaterals are not sufficient and the tissue clock has started. Published series of Gartland III fractures report acute ischaemia or a pink pulseless hand in roughly one in six, so the distinction is a routine clinical decision rather than an academic one, and perfusion must be reassessed and documented after reduction.

Because in a frail older patient the danger comes from lying still, not from the broken bone. An unfixed hip fracture is intensely painful on any movement, so the patient cannot sit up, cannot cough effectively, cannot be turned comfortably and cannot be mobilised. Within a day or two that produces atelectasis and aspiration risk, pressure injury over the sacrum and heels, venous stasis, dehydration and constipation from opioid analgesia, and delirium driven by pain, immobility and unfamiliar surroundings. Each of these is itself a predictor of death, and they compound. Fixation is best understood as the intervention that allows nursing and mobilisation to begin, rather than as the treatment of the fracture alone. Meta-analysis of over 190,000 patients found roughly 20 per cent lower one-year mortality with surgery within 48 hours, and this is why hip fracture now runs as a time-critical pathway with orthogeriatric co-care, pre-operative optimisation done in parallel rather than in series, and audit of time to theatre as a quality measure.

For two separate reasons that happen to coincide. The first is articular. Types III and IV cross the epiphysis and therefore break the joint surface, and cartilage cannot remodel a step, so any residual incongruity becomes post-traumatic arthritis in a joint that has to last a lifetime. The second is the physis itself. In types I and II the fracture line runs along the plane of the physis and exits through the metaphysis, which is relatively forgiving because the germinal layer of the plate stays with the epiphysis. In types III and IV the line crosses the physis vertically, so the germinal layer is split and the metaphyseal and epiphyseal bone lie directly against each other. If they heal in contact, a bony bar forms across the plate and growth stops locally. Anatomical reduction restores the barrier between them and reduces the chance of a bar. The technical corollary is that fixation should use screws placed parallel to the physis within the epiphysis or within the metaphysis, avoiding hardware crossing the plate, precisely because crossing it risks creating the tether the operation exists to prevent.
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